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The general situations of the eye were observed and the corneal thickness were measured with ultrasonic corneal pachymeter after the animal models was established. After a week, the corneas were removed after the experimental animals are put to death. The corneal endothelial cell density of 12 half-chip were counted through Alizarin Red-Trypan blue staining; 12 half-clip corneas were fixed with 4% neutral formalin solution , HE staining was performed, the expression of AQP-1 in corneal stroma and corneal endothelial cell were detected through immunohistochemical staining; Na~+-K~+-ATPase activities in 12 half-clip corneas were examined with Na~+-K~+-ATPase kit; the expression of AQP-1 mRNA were detected through real-time fluorescent quantitation PCR.

术后观察眼球大体情况、测量角膜厚度。1周后处死实验动物取角膜，用茜素红-台盼蓝染色染色行角膜内皮细胞计数；用4%中性福尔马林溶液固定行HE染色、应用免疫组化染色检测AQP-1在角膜基质和内皮细胞表达的改变；用Na~+-K~+-ATP酶试剂盒测量角膜内皮细胞Na~+-K~+-ATP酶活性；实时荧光定量PCR检测AQP-1mRNA在角膜内皮细胞表达的改变；并于正常对照组角膜比较。

To explore (1)whether the EPO is present in gastric carcinoma ;(2)the possible mechanism of its expression;(3) the role of EPO in tumor angiogenesis and its results, we investigated the expression of EPO, HIF-la in gastric carcinoma and marked EC with CD105( monoclonal antibody MS1290 )and CD34 on paraffinic slides. Analysis of the realationship between EPO MVDand HIF-la in human gastric carcinoma.

本实验检测人胃癌及癌旁组织中EPO、HIF-1a的表达，同时进行了小鼠抗人CD105单克隆抗体(MS-1290)标记和CD34标记，分析EPO与微血管密度及HIF-1a表达的关系，以期明了①胃癌组织中是否存在EPO的表达、②表达的途径和机制、③EPO与肿瘤血管生成的关系和可能的后果。

PDJ also could induce expression of PRP, and this trend was significant when the tobacco seedlings were infected by Phytophthora parasitica Var nicotianae. The results suggested that the increased activities of these enzymes and the expression of PRP is most likely to be associated with the development of induced resistance in tobacco seedlings.

PDJ处理同样诱导了烟草体内某些病程相关蛋白的表达，并且在黑胫病菌侵染后表达增强，这些结果表明PDJ处理后烟草酶活性的变化和病程相关蛋白的表达可能与其诱发烟草幼苗系统获得抗性的表现有关。

AIM: To investigate the expression of PTEN and P16 protein expression as well as their correlations with the pathobiological behaviors and prognosis of PGC.

目的：探讨PTEN、P16在原发性胆囊癌中的表达与其病理生物学行为和预后的关系。

Abnormal expression of PTEN gene was involved in progression of ovarian cancer probably by decreasing cell adhesion and apoptosis, increasing cell mobility and angiogenesis. Reduced expression of PTEN gene was closely associated with tumorigenesis and pathobiological behavior of ovarian endometrioid cancer.

PTEN蛋白表达下调与卵巢恶性肿瘤的组织分化程度、临床病理分期和转移能力相关，与卵巢癌中的子宫内膜样癌PTEN蛋白表达关系更为密切。

In Hp positive group, the expression of IL-10 in active gastritis is lower than that in inactive gastritis, which are different with the expression of COX-2 and iNOS, indicating IL-10 is a protect factor in gastritis, but COX-2 and iNOS may relate to pathopoiesis.

Hp阳性组，IL-10在活动性胃炎粘膜组织中的表达低于非活动性胃炎，COX-2、iNOS在活动性胃炎中的表达显著高于非活动性胃炎，说明IL-10在活动性胃炎的发病中可能起保护作用，而COX-2、iNOS可能与致病过程有关。

Hewever the caspase-3 expression was still very high in cortex. Conclusion: In KA model the hippocampus amygdala plays a key role in epilepsy. Therefore it provides a mimic optimal conditions for human temporal lobe epilepsy study. Its an effective method of studying the development, change and pathopoiesis of epilepsy. There is a close relationship between NPY and epilepsy. The regulation of NPY via VPA may be one of the antiepilepsy mechanism of mechanism of VPA. Moreover NGF can enhance the caspase-3 expression. Excito-amino acid can aggravate the apoptosis, which may be one of the reason for intractability of epilepsy.

KA模型因其海马杏仁核在癫痈中起核心作用，故具有模拟人类颗叶癫痈的理想条件，是研究癫痈发展变化、致痈机理的有效工具，NPY与癫病有着密切的联系，VPA调节NPY有可能是其抗癫痈机理之一，而NGF对caspase一3表达有促进作用，兴奋性氨基酸可造成细胞凋亡的增加，而凋亡细胞的增多有可能是癫痈久治不愈的原因之一。

Results］SVHRP could obviously delay forming a time of seizure susceptibility, and decrease the expression of PENK compared with KA leads to increase the expression of PENK.

结果］蝎毒耐热蛋白干预组的大鼠较单独给予红藻氨酸组的大鼠癫痫敏感性形成率明显降低（P＜0.05），而且前脑啡肽原的表达也明显下降（P＜0.05）。

(1) NSCLC has a higher expression of F1ATPase-α and the expression is on the pericellular membrane of A549 cells, which may provide a new target for molecular therapy of NSCLC.

2F1ATPase-α表达于非小细胞肺癌A549细胞株的细胞膜上，提示有作为靶向药物治疗分子靶点的潜力。

This pattern is characterized by the combination of pro-MMP-2 and membrane type 1 (MT1)-MMP expression, which drie pericellular generation of actie MMP-2 and local degradation of normal lier matrix. In addition there is a marked increase in expression of TIMP-1 leading to a more global inhibition of degradation of fibrillar lier collagens by interstitial collagenases (MMP-1/MMP-13). These pathways play a significant role in the progression of lier fibrosis.

其降解肝正常基质，同时抑制引起肝纤维化的纤维胶原降解，这种情况的特点是前明胶酶A合成和膜型1－MMP表达，使得小叶内细胞激活MMP-2和降解当处的正常肝基质，此外血浆1型组织基质金属蛋白酶抑制剂（TIMP-1）明显升高引起广泛的抑制间隙胶原酶(MMP-1/MMP-13)对纤维肝脏胶原的降解。

"Second Life is remarkably easy to work with, and is very popular,"

"第二次生命是显着容易的工作，并且很受欢迎，"

For example, we usually assume that materials are homogeneous and isotropic and free of internal defects or flaws.

为了得到适合有限元分析的模型，我们必须经过如图2所示的简化步骤。